By Gaspare De Fiore
Wanted N°4 and N°5 ; Has an individual bought it?
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Extra resources for Corso di disegno 2
5) Establish and characterize a new experimental system to test the mitogenic and oncogenic potential of the hLHR. ; Professor; Medicine; Emory University 1784 North Decatur Road Atlanta, Ga 30322 Timing: Fiscal Year 2002; Project Start 01-AUG-2000; Project End 31-JUL-2004 Summary: Although studies from a variety of species indicate that a structural or functional shift in neurochemical input into gonadotropin releasing hormone (GnRH) neurons induces the developmental increase in GnRH secretion initiating puberty, the factor(s) responsible for this change are not known.
In addition, we presented evidence suggesting that FSH also plays an important role in LH-mediated ovarian androgen production. Although KAL1 and GNRHR comprise about 20% of the mutations described to date, the molecular basis for most IHH patients remains unknown. The long-term goal of our laboratory is to advance our understanding of normal puberty and reproduction by determining the molecular basis of IHH. Our overlying hypothesis is that the genes that regulate gonadotropin production and/or secretion are important in normal puberty and possess mutations in IHH patients.
The first global aim is to investigate the anabolic actions of parathyroid hormone (PTH) on the skeleton, particularly as an agent to prevent estrogen deficiency bone loss. There are 2 major projects to address this aim: 1) an investigation of the effects of ability of short-term parathyroid hormone administration on bone loss in young women with GnRH analog-induced estrogen deficiency and 2) an investigation of the effects of ability of long-term parathyroid hormone administration to prevent bone loss in early postmenopausal women.